Manifestation of psychiatric behaviors in a mouse model of griseofulvin-induced hepatic porphyria
Author:
Affiliation:
1. Faculty of Pharmaceutical Sciences, Josai International University
2. Faculty of Pharmaceutical Sciences, Josai University
Publisher
Japanese Society of Toxicology
Subject
Toxicology
Link
http://www.jstage.jst.go.jp/article/jts/33/5/33_5_599/_pdf
Reference39 articles.
1. Anderson, K.E., Bloomer, J.R., Bonkovsky, H.L., Kushner, J.P., Pierach, C.A., Pimstone, N.R. and Desnick, R.J. (2005): Recommendations for the diagnosis and treatment of the acute porphyrias. Ann. Intern. Med., 142, 439-450.
2. Berenson, M.M., Welch, V. and Garcia-Marin, J.J. (1991): Importance of bile acid structure in amelioration of griseofulvin-induced murine protoporphyric hepatopathy. J. Lab. Clin. Med., 118, 89-98.
3. Billi de Catabbi, S.C., Faletti, A., Fuentes, F., San Martín de Viale, L.C. and Cochón, A.C. (2005): Hepatic arachidonic acid metabolism is disrupted after hexachlorobenzene treatment. Toxicol. Appl. Pharmacol., 204, 187-195.
4. Bonkowsky, H.L. and Schady, W. (1982): Neurologic manifestations of acute porphyria. Semin. Liver Dis., 2, 108-124.
5. Hepatic protoporphyria is associated with a decrease in ligand binding for the mitochondrial benzodiazepine receptors in the liver
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