Role of c-Jun N-terminal kinase (JNK) in regulating tumor necrosis factor-alpha (TNF-.ALPHA.) mediated increase of acetaminophen (APAP) and chlorpromazine (CPZ) toxicity in murine hepatocytes-Reference-Cited by-同舟云学术

Role of c-Jun N-terminal kinase (JNK) in regulating tumor necrosis factor-alpha (TNF-.ALPHA.) mediated increase of acetaminophen (APAP) and chlorpromazine (CPZ) toxicity in murine hepatocytes

Published:2010 Issue:2 Volume:35 Page:163-173
ISSN:0388-1350
Container-title:The Journal of Toxicological Sciences
language:en
Short-container-title:J. Toxicol. Sci.

Author:

Gandhi Adarsh¹,Guo Tao¹,Ghose Romi¹

Affiliation:

1. University of Houston, Department of Pharmacological and Pharmaceutical Sciences, College of Pharmacy

Publisher

Japanese Society of Toxicology

Subject

Toxicology

Link

http://www.jstage.jst.go.jp/article/jts/35/2/35_2_163/_pdf

Reference41 articles.

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2. Blazka, M.E., Elwell, M.R., Holladay, S.D., Wilson, R.E. and Luster, M.I. (1996): Histopathology of acetaminophen-induced liver changes: role of interleukin 1 alpha and tumor necrosis factor alpha. Toxicol. Pathol., 24, 181-189.

3. Role of Proinflammatory Cytokines in Acetaminophen Hepatotoxicity

4. Acetaminophen hepatotoxicity in tumor necrosis factor/lymphotoxin-? gene knockout mice

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