Hepatic glutathione contributes to attenuation of thioacetamide-induced hepatic necrosis due to suppression of oxidative stress in diet-induced obese mice
Author:
Affiliation:
1. Medicinal Safety Research Laboratories, Daiichi Sankyo Co., Ltd.
Publisher
Japanese Society of Toxicology
Subject
Toxicology
Link
https://www.jstage.jst.go.jp/article/jts/40/4/40_509/_pdf
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2. Alberici, L.C., Vercesi, A.E. and Oliveira, H.C. (2011): Mitochondrial energy metabolism and redox responses to hypertriglyceridemia. J. Bioenerg. Biomembr., 43, 19-23.
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4. Chilakapati, J., Shankar, K., Korrapati, M.C., Hill, R.A. and Mehendale, H.M. (2005): Saturation toxicokinetics of thioacetamide: role in initiation of liver injury. Drug Metab. Dispos., 33, 1877-1885.
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3. Cilostazol attenuates indices of liver damage induced by thioacetamide in albino rats through regulating inflammatory cytokines and apoptotic biomarkers;European Journal of Pharmacology;2018-03
4. Decreased hepatic phosphorylated p38 mitogen-activated protein kinase contributes to attenuation of thioacetamide-induced hepatic necrosis in diet-induced obese mice;The Journal of Toxicological Sciences;2016
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