Maternal exposure to dioxin reduces hypothalamic but not pituitary metabolome in fetal rats: a possible mechanism for a fetus-specific reduction in steroidogenesis
Author:
Affiliation:
1. Graduate School of Pharmaceutical Sciences, Kyushu University
2. Innovation Center for Medical Redox Navigation, Kyushu University
3. Graduate School of Agricultural Sciences, Kyushu University
Publisher
Japanese Society of Toxicology
Subject
Toxicology
Link
http://www.jstage.jst.go.jp/article/jts/35/3/35_3_365/_pdf
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3. Buerstatte, C.R., Behar, K.L., Novotny, E.J. and Lai, J.C. (2000): Brain regional development of the activity of alpha-ketoglutarate dehydrogenase complex in the rat. Brain Res. Dev. Brain Res., 125, 139-145.
4. In Utero and Lactational Exposure of Female Holtzman Rats to 2,3,7,8- Tetrachlorodibenzo-p-Dioxin: Modulation of the Estrogen Signal1
5. Hays, L.E., Carpenter, C.D. and Petersen, S.L. (2002): Evidence that GABAergic neurons in the preoptic area of the rat brain are targets of 2,3,7,8-tetrachlorodibenzo-p-dioxin during development. Environ. Health Perspect., 110 (Suppl. 3), 369-376.
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