1. Block, M.L., Wu, X., Pei, Z., Li, G., Wang, T., Qin, L., Wilson, B., Yang, J., Hong, J.S. and Veronesi, B. (2004): Nanometer size diesel exhaust particles are selectively toxic to dopaminergic neurons: the role of microglia, phagocytosis, and NADPH oxidase. FASEB J, 18, 1618-1620.
2. Calderón-Garcidueñas, L., Maronpot, R.R., Torres-Jardon, R., Henríquez-Roldán, C., Schoonhoven, R., Acuña-Ayala, H., Villarreal-Calderón, A., Nakamura, J., Fernando, R., Reed, W., Azzarelli, B. and Swenberg, J.A. (2003): DNA damage in nasal and brain tissues of canines exposed to air pollutants is associated with evidence of chronic brain inflammation and neurodegeneration. Toxicol. Pathol., 31, 524-538.
3. Calderón-Garcidueñas, L., Mora-Tiscareño, A., Ontiveros, E., Gómez-Garza, G., Barragán-Mejía, G., Broadway, J., Chapman, S., Valencia-Salazar, G., Jewells, V., Maronpot, R.R., Henríquez-Roldán, C., Pérez-Guillé, B., Torres-Jardón, R., Herrit, L., Brooks, D., Osnaya-Brizuela, N., Monroy, M.E., González-Maciel, A., Reynoso-Robles, R., Villarreal-Calderon, R., Solt, A.C. and Engle, R.W. (2008a): Air pollution, cognitive deficits and brain abnormalities: a pilot study with children and dogs. Brain Cogn, 68, 117-127.
4. Calderón-Garcidueñas, L., Reed, W., Maronpot, R.R., Henríquez- Roldán, C., Delgado-Chavez, R., Calderón-Garcidueñas, A., Dragustinovis, I., Franco-Lira, M., Aragón-Flores, M., Solt, A.C., Altenburg, M., Torres-Jardón, R. and Swenberg, J.A. (2004): Brain inflammation and Alzheimer’s-like pathology in individuals exposed to severe air pollution. Toxicol. Pathol., 32, 650-658.
5. Calderón-Garcidueñas, L., Solt, A.C., Henríquez-Roldán, C., Torres-Jardón, R., Nuse, B., Herritt, L., Villarreal-Calderón, R., Osnaya, N., Stone, I., García, R., Brooks, D.M., González-Maciel, A., Reynoso-Robles, R., Delgado-Chávez, R. and Reed, W. (2008b): Long-term air pollution exposure is associated with neuroinflammation, an altered innate immune response, disruption of the blood-brain barrier, ultrafine particulate deposition, and accumulation of amyloid beta-42 and alpha-synuclein in children and young adults. Toxicol. Pathol., 36, 289-310.