Neonatal exposure to 2,3,7,8-tetrachlorodibenze-p-dioxin increases the mRNA expression of prostatic proteins in C57BL mice
Author:
Affiliation:
1. Endocrine Research Group, Department of Disease Model, Research Institute for Radiation Biology and Medicine, Hiroshima University
2. Division of Toxicology, National Institute of Health Sciences, Japan
Publisher
Japanese Society of Toxicology
Subject
Toxicology
Link
https://www.jstage.jst.go.jp/article/jts/38/2/38_279/_pdf
Reference16 articles.
1. Bjerke, D.L. and Peterson, R.E. (1994): Reproductive toxicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin in male rats: different effects of in utero versus lactational exposure. Toxicol. Appl. Pharmacol., 127, 241-249.
2. Cunha, G.R., Donjacour, A.A., Cooke, P.S., Mee, S., Bigsby, R.M., Higgins, S.J. and Sugimura, Y. (1987): The endocrinology and developmental biology of the prostate. Endoc. Rev., 8, 338-362.
3. Fujimoto, N., Akimoto, Y., Suzuki, T., Kitamura, S. and Ohta, S. (2006): Identification of prostatic-secreted proteins in mice by mass spectrometric analysis and evaluation of lobe-specific and androgen-dependent mRNA expression. J. Endo., 190, 793-803.
4. Kamada, M., Mori, H., Maeda, N., Yamamoto, S., Kunimi, K., Takikawa, M., Maegawa, M., Aono, T., Futaki, S. and Koide, S.S. (1998): beta-Microseminoprotein/prostatic secretory protein is a member of immunoglobulin binding factor family. Biochem. Biophys. Acta, 1388, 101-110.
5. Ko, K., Theobald, H.M. and Moore, R.W. and Peterson, R.E. (2004): Evidence that inhibited prostatic epithelial bud formation in 2,3,7,8-tetrachlorodibenzo-p-dioxin-exposed C57BL/6J fetal mice is not due to interruption of androgen signaling in the urogenital sinus. Toxic. Sci., 79, 360-369.
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1. Animal Toxicology Studies on the Male Reproductive Effects of 2,3,7,8-Tetrachlorodibenzo-p-Dioxin: Data Analysis and Health Effects Evaluation;Frontiers in Endocrinology;2021-11-03
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