A monkey model of acetaminophen-induced hepatotoxicity; phenotypic similarity to human
Author:
Affiliation:
1. Medicinal Safety Research Laboratories, Daiichi Sankyo Co., Ltd.
2. R&D Planning & Management Department, Daiichi Sankyo Co., Ltd.
Publisher
Japanese Society of Toxicology
Subject
Toxicology
Link
https://www.jstage.jst.go.jp/article/jts/42/1/42_73/_pdf
Reference44 articles.
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3. Dahlin, D.C., Miwa, G.T., Lu, A.Y. and Nelson, S.D. (1984): N-acetyl-p-benzoquinone imine: a cytochrome P-450-mediated oxidation product of acetaminophen. Proc. Natl. Acad. Sci. USA, 81, 1327-1331.
4. Dai, G., He, L., Chou, N. and Wan, Y.J. (2006): Acetaminophen metabolism does not contribute to gender difference in its hepatotoxicity in mouse. Toxicol. Sci., 92, 33-41.
5. Davis, D.C., Potter, W.Z., Jollow, D.J. and Mitchell, J.R. (1974): Species differences in hepatic glutathione depletion, covalent binding and hepatic necrosis after acetaminophen. Life Sci., 14, 2099-2109.
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