Dihydropyrazine induces endoplasmic reticulum stress and inhibits autophagy in HepG2 human hepatoma cells
Author:
Affiliation:
1. Faculty of Pharmaceutical Sciences, Sojo University
2. School of Pharmacy at Fukuoka, International University of Health and Welfare
Publisher
Japanese Society of Toxicology
Link
https://www.jstage.jst.go.jp/article/jts/49/7/49_313/_pdf
Reference34 articles.
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2. Esaki, M., Ishida, T., Miyauchi, Y. and Takechi, S. (2020): The effect of dihydropyrazines on lipopolysaccharide-stimulated human hepatoma HepG2 cells via regulating the TLR4-MyD88-mediated NF-κB signaling pathway. J. Toxicol. Sci., 45, 401-409.
3. Hanada, T., Noda, N.N., Satomi, Y., Ichimura, Y., Fujioka, Y., Takao, T., Inagaki, F. and Ohsumi, Y. (2007): The Atg12-Atg5 conjugate has a novel E3-like activity for protein lipidation in autophagy. J. Biol. Chem., 282, 37298-37302.
4. Harding, H.P., Novoa, I., Zhang, Y., Zeng, H., Wek, R., Schapira, M. and Ron, D. (2000): Regulated translation initiation controls stress-induced gene expression in mammalian cells. Mol. Cell, 6, 1099-1108.
5. Ishida, T., Takechi, S. and Yamaguchi, T. (2012): Possible involvement of glutathione balance disruption in dihydropyrazine-induced cytotoxicity on human hepatoma HepG2 cells. J. Toxicol. Sci., 37, 1065-1069.
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