2,3,7,8-Tetrachlorodibenzo-p-dioxin potentially attenuates the gene expression of pituitary gonadotropin .BETA.-subunits in a fetal age-specific fashion: a comparative study using cultured pituitaries
Author:
Affiliation:
1. Graduate School of Pharmaceutical Sciences, Kyushu University
2. Faculty of Pharmaceutical Sciences, Nagasaki International University
3. Faculty of Pharmaceutical Sciences, Sojo University
Publisher
Japanese Society of Toxicology
Subject
Toxicology
Link
http://www.jstage.jst.go.jp/article/jts/36/2/36_2_221/_pdf
Reference41 articles.
1. Apfelbaum, M.E. and Taleisnik, S. (1976): Interaction between oestrogen and gonadotrophin-releasing hormone on the release and synthesis of luteinizing hormone and follicle-stimulating hormone from incubated pituitaries. J. Endocrinol., 68, 127-136.
2. Aubert, M.L., Begeot, M., Winiger, B.P., Morel, G., Sizonenko, P.C. and Dubois, P.M. (1985): Ontogeny of hypothalamic luteinizing hormone-releasing hormone (GnRH) and pituitary GnRH receptors in fetal and neonatal rats. Endocrinology, 116, 1565-1576.
3. Badawi, A.F., Cavalieri, E.L. and Rogan, E.G. (2000): Effect of chlorinated hydrocarbon on expression of cytochrome P450 1A1, 1A2 and 1B1 and 2-and 4-hydroxylation of 17β-estradiol in female Sprague-Dawley rats. Carcinogenesis, 21, 1593-1599.
4. Comparative in vitro and in vivo protein kinase C activation by selected pesticides and transition metal salts
5. Altered regulation of pituitary gonadotropin-releasing hormone (GnRH) receptor number and pituitary responsiveness to GnRH in 2,3,7,8-tetrachlorodibenzo-p-dioxin-treated male rats
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