Normal ovarian aging, but modified T-cell differentiation, in female mice following neonatal exposure to bisphenol A
Author:
Affiliation:
1. Hatano Research Institute, Food and Drug Safety Center
Publisher
Japanese Society of Toxicology
Link
https://www.jstage.jst.go.jp/article/fts/4/1/4_15/_pdf
Reference25 articles.
1. Adewale, H.B., Jefferson, W.N., Newbold, R.R. and Patisaul, H.B. (2009): Neonatal bisphenol-a exposure alters rat reproductive development and ovarian morphology without impairing activation of gonadotropin-releasing hormone neurons. Biol. Reprod., 81, 690-699.
2. Ashby, J., Owens, W., Odum, J. and Tinwell, H. (2003): The intact immature rodent uterotrophic bioassay: possible effects on assay sensitivity of vomeronasal signals from male rodents and strain differences. Environ. Health Perspect., 111, 1568-1570.
3. Gameiro, C.M., Romão, F. and Castelo-Branco, C. (2010): Menopause and aging: changes in the immune system--a review. Maturitas, 67, 316-320.
4. Ghosh, M., Rodriguez-Garcia, M. and Wira, C.R. (2014): The immune system in menopause: pros and cons of hormone therapy. J. Steroid Biochem. Mol. Biol., 142, 171-175.
5. Honma, S., Suzuki, A., Buchanan, D.L., Katsu, Y., Watanabe, H. and Iguchi, T. (2002): Low dose effect of in utero exposure to bisphenol A and diethylstilbestrol on female mouse reproduction. Reprod. Toxicol., 16, 117-122.
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