Cigarette smoke extract-induced interleukin-6 expression is regulated by phospholipase D1 in human bronchial epithelial cells
Author:
Affiliation:
1. Biomedical Research Institute and Department of Biochemistry & Molecular Biology, College of Medicine, Hanyang University, Korea
Publisher
Japanese Society of Toxicology
Subject
Toxicology
Link
https://www.jstage.jst.go.jp/article/jts/41/1/41_77/_pdf
Reference66 articles.
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2. Birnbaumer, L. (2007): Expansion of signal transduction by G proteins. The second 15 years or so: from 3 to 16 alpha subunits plus betagamma dimers. Biochim. Biophys. Acta, 1768, 772-793.
3. Boudreau, R.T., Garduno, R. and Lin, T.J. (2002): Protein phosphatase 2A and protein kinase Calpha are physically associated and are involved in Pseudomonas aeruginosa-induced interleukin 6 production by mast cells. J. Biol. Chem., 277, 5322-5329.
4. Bozinovski, S., Seow, H.J., Chan, S.P., Anthony, D., McQualter, J., Hansen, M., Jenkins, B.J., Anderson, G.P. and Vlahos, R. (2015): Innate cellular sources of interleukin-17A regulate macrophage accumulation in cigarette- smoke-induced lung inflammation in mice. Clin. Sci. (Lond), 129, 785-796.
5. Brandenburg, L.O., Pufe, T. and Koch, T. (2014): Role of phospholipase d in g-protein coupled receptor function. Membranes, 4, 302-318.
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