Arsenite exposure potentiates apoptosis-inducing effects of tumor necrosis factor-alpha- through reactive oxygen species

Author:

Singhirunnusorn Pattama1,Moolmuang Benchamart1,Lirdprapamongkol Kriengsak2,Ruchirawat Mathuros3

Affiliation:

1. Laboratory of Chemical Carcinogenesis, Chulabhorn Research Institute, Thailand

2. Laboratory of Biochemistry, Chulabhorn Research Institute, Thailand

3. Laboratory of Environmental Toxicology, Chulabhorn Research Institute, Thailand

Publisher

Japanese Society of Toxicology

Subject

Toxicology

Reference38 articles.

1. Aguirre-Bañuelos, P., Escudero-Lourdes, C., Sanchez-Peña, L.C., Del Razo, L.M. and Perez-Urizar, J. (2008): Inorganic arsenic exposure affects pain behavior and inflammatory response in rat. Toxicol. Appl. Pharmacol., 229, 374-385.

2. Blaser, H., Dostert, C., Mak, T.W. and Brenner, D. (2016): TNF and ROS Crosstalk in Inflammation. Trends Cell Biol., 26, 249-261.

3. Cappellini, A., Tazzari, P.L., Mantovani, I., Billi, A.M., Tassi, C., Ricci, F., Conte, R. and Martelli, A.M. (2005): Antiapoptotic role of p38 mitogen activated protein kinase in Jurkat T cells and normal human T lymphocytes treated with 8-methoxypsoralen and ultraviolet-A radiation. Apoptosis, 10, 141-152.

4. Chang, L. and Karin, M. (2001): Mammalian MAP kinase signalling cascades. Nature, 410, 37-40.

5. Dagenais, M., Douglas, T. and Saleh, M. (2014): Role of programmed necrosis and cell death in intestinal inflammation. Curr. Opin. Gastroenterol., 30, 566-575.

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