Hypothyroidism protects di(n-butyl) phthalate-induced reproductive organs damage in Sprague-Dawley male rats
Author:
Affiliation:
1. College of Pharmacy, Pusan National University
2. Division of Toxicology, College of Pharmacy, Sungkyunkwan University
3. National Institute of Toxicological Research, Korea Food and Drug Administration
Publisher
Japanese Society of Toxicology
Subject
Toxicology
Link
http://www.jstage.jst.go.jp/article/jts/33/3/33_3_299/_pdf
Reference30 articles.
1. Brucker-Davis, F. (1998): Effects of environmental synthetic chemicals on thyroid function. Thyroid, 8, 827-856.
2. Choksi, N.Y., Jahnke, G.D., St Hilaire, C. and Shelby, M. (2003): Role of thyroid hormones in human and laboratory animal reproductive health. Birth Defects Res. B. Dev. Reprod. Toxicol., 68, 479-491.
3. Thyroid Hormones and Testis Development: A Model System for Increasing Testis Growth and Sperm Production
4. Cooke, P.S. and Meisami, E. (1991): Early hypothyroidism in rats causes increased adult testis and reproductive organ size but does not change testosterone levels. Endocrinology, 129, 237-243.
5. Gaitan, E. (1983): Endemic goiter in western Colombia. Ecol. Dis., 2, 295-308.
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2. Di-n-butyl phthalate prompts interruption of spermatogenesis, steroidogenesis, and fertility associated with increased testicular oxidative stress in adult male rats;Environmental Science and Pollution Research;2017-06-24
3. A crossover–crossback prospective study of dibutyl-phthalate exposure from mesalamine medications and semen quality in men with inflammatory bowel disease;Environment International;2016-10
4. Effects and Predicted Consequences of Persistent and Bioactive Organic Pollutants on Thyroid Function;Effects of Persistent and Bioactive Organic Pollutants on Human Health;2013-07-26
5. Adult-Onset Hypothyroidism Enhances Fear Memory and Upregulates Mineralocorticoid and Glucocorticoid Receptors in the Amygdala;PLoS ONE;2011-10-24
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