Nitric Oxide(NO) Primarily Accounts for Endothelium-Dependent Component of .BETA.-Adrenoceptor-Activated Smooth Muscle Relaxation of Mouse Aorta in Response to Isoprenaline.-Reference-Cited by-同舟云学术

Nitric Oxide(NO) Primarily Accounts for Endothelium-Dependent Component of .BETA.-Adrenoceptor-Activated Smooth Muscle Relaxation of Mouse Aorta in Response to Isoprenaline.

Published:2002 Issue:4/5 Volume:38 Page:87-99
ISSN:0916-8737
Container-title:Journal of Smooth Muscle Research
language:en
Short-container-title:J. Smooth Muscle Res.

Author:

Akimoto Yurie¹,Horinouchi Takahiro¹,Shibano Mari¹,Matsushita Mayumi¹,Yamashita Yoko¹,Okamoto Takao¹,Yamaki Fumiko¹,Tanaka Yoshio¹,Koike Katsuo¹

Affiliation:

1. Department of Chemical Pharmacology, Toho University School of Pharmaceutical Sciences

Publisher

Japan Society of Smooth Muscle Research

Subject

General Medicine,Physiology

Link

http://www.jstage.jst.go.jp/article/jsmr/38/4,5/38_4,5_87/_pdf

Reference22 articles.

1. Role of endothelium/nitric oxide in atypical β-adrenoceptor-mediated relaxation in rat isolated aorta

2. Vasorelaxant response to isoprenaline, nitric oxide donor, calcitonin gene-related peptide and vasoactive intestinal peptide in aortic rings of adult C57BL/6J mice

3. Acetylcholine releases endothelium-derived hyperpolarizing factor and EDRF from rat blood vessels

4. Endothelium-dependent relaxation of coronary arteries by noradrenaline and serotonin

5. Effects of cyclic GMP elevation on isoprenaline-induced increase in cyclic AMP and relaxation in rat aortic smooth muscle: role of phosphodiesterase 3

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