Mitotic recombination and compound-heterozygous mutations are predominant NF1-inactivating mechanisms in children with juvenile myelomonocytic leukemia and neurofibromatosis type 1-Reference-Cited by-同舟云学术

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Mitotic recombination and compound-heterozygous mutations are predominant NF1-inactivating mechanisms in children with juvenile myelomonocytic leukemia and neurofibromatosis type 1

Published:2009-12-16 Issue:2 Volume:95 Page:320-323
ISSN:0390-6078
Container-title:Haematologica
language:en
Short-container-title:Haematologica

Author:

Steinemann D.,Arning L.,Praulich I.,Stuhrmann M.,Hasle H.,Stary J.,Schlegelberger B.,Niemeyer C. M.,Flotho C.

Publisher

Ferrata Storti Foundation (Haematologica)

Subject

Hematology

Cited by 55 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Cytogenetics and genomics in CML and other myeloproliferative neoplasms;Best Practice & Research Clinical Haematology;2024-06

2. Germline Predisposition to Myeloid Neoplasms;Clinics in Laboratory Medicine;2023-12

3. Evaluation of Molecular and Clinical Findings in Children With Neurofibromatosis Type 1: Identification of 15 Novel Variants;Pediatric Neurology;2023-12

4. Biallelic inactivation of the NF1 tumour suppressor gene in juvenile myelomonocytic leukaemia: Genetic evidence of driver function and implications for diagnostic workup;British Journal of Haematology;2023-11-09

5. The association between juvenile xanthogranulomas in neurofibromatosis type 1 patients and the development of leukaemia: A systematic review;Journal of the European Academy of Dermatology and Venereology;2023-07-20

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