APOL1 , α-thalassemia, and BCL11A variants as a genetic risk profile for progression of chronic kidney disease in sickle cell anemia-Reference-Cited by-同舟云学术

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APOL1 , α-thalassemia, and BCL11A variants as a genetic risk profile for progression of chronic kidney disease in sickle cell anemia

Published:2016-09-22 Issue:1 Volume:102 Page:e1-e6
ISSN:0390-6078
Container-title:Haematologica
language:en
Short-container-title:Haematologica

Author:

Saraf Santosh L.,Shah Binal N.,Zhang Xu,Han Jin,Tayo Bamidele O.,Abbasi Taimur,Ostrower Adam,Guzman Elizabeth,Molokie Robert E.,Gowhari Michel,Hassan Johara,Jain Shivi,Cooper Richard S.,Machado Roberto F.,Lash James P.,Gordeuk Victor R.

Publisher

Ferrata Storti Foundation (Haematologica)

Subject

Hematology

Cited by 48 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. A moonlighting job for α-globin in blood vessels;Blood;2024-08-22

2. Relevance of repeated analyses of albuminuria and glomerular filtration rate in African children with sickle cell anaemia;British Journal of Haematology;2024-06-27

3. Systematic Review of Genetic Modifiers Associated with the Development and/or Progression of Nephropathy in Patients with Sickle Cell Disease;International Journal of Molecular Sciences;2024-05-16

4. Prevalence of kidney health genetic variants in adults with sickle cell nephropathy;British Journal of Haematology;2024-05-12

5. Les complications de la drépanocytose, de l’enfant à l’adulte;Bulletin de l'Académie Nationale de Médecine;2024-05

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