SDF1α-induced chemotaxis of JAK2-V617F-positive cells is dependent on Bruton tyrosine kinase and its downstream targets PI3K/AKT, PLCγ1 and RhoA-Reference-Cited by-同舟云学术

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SDF1α-induced chemotaxis of JAK2-V617F-positive cells is dependent on Bruton tyrosine kinase and its downstream targets PI3K/AKT, PLCγ1 and RhoA

Published:2019-02-14 Issue:7 Volume:104 Page:e288-e292
ISSN:0390-6078
Container-title:Haematologica
language:en
Short-container-title:Haematologica

Author:

Nimmagadda Subbaiah Chary,Frey Stephanie,Müller Peter,Wolleschak Denise,Weinert Sönke,Keller Ulrich,Edelmann Bärbel,Fischer Thomas

Publisher

Ferrata Storti Foundation (Haematologica)

Subject

Hematology

Cited by 5 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Understanding Aberrant Signaling to Elude Therapy Escape Mechanisms in Myeloproliferative Neoplasms;Cancers;2022-02-15

2. Stromal Cell-Derived Factor-1α (SDF-1α) Promotes Growth and Migration of Bone Marrow Stromal Cells (BMSCs) and Gastric Cancer Cells Through Phosphatidylinositol 3-Kinase/AKT (PI3K/Akt) Pathway;Journal of Biomaterials and Tissue Engineering;2022-02-01

3. Anti-inflammatory treatment in MPN: targeting TNFR1 and TNFR2 in JAK2-V617F–induced disease;Blood Advances;2021-12-09

4. Application of Single-Cell Approaches to Study Myeloproliferative Neoplasm Biology;Hematology/Oncology Clinics of North America;2021-04

5. Bone marrow niches in haematological malignancies;Nature Reviews Cancer;2020-02-28

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