ER-Mitochondria Communication. How Privileged?-Reference-Cited by-同舟云学术

ER-Mitochondria Communication. How Privileged?

Published:2007-08 Issue:4 Volume:22 Page:261-268
ISSN:1548-9213
Container-title:Physiology
language:en
Short-container-title:Physiology

Author:

Franzini-Armstrong Clara¹

Affiliation:

1. Department of Cell and Developmental Biology, School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania,

Abstract

Mitochondria have a low affinity for Ca2+, but they take up these ions during normal cell activity because they are in close proximity to the sites of calcium entry into the cell and of internal Ca2+release. This gives mitochondria privileged access to cytoplasmic Ca2+without requiring a direct communication with the endoplasmic reticulum.

Publisher

American Physiological Society

Subject

Physiology

Link

https://www.physiology.org/doi/pdf/10.1152/physiol.00017.2007

Reference76 articles.

1. Mitochondrial oversight of cellular Ca2+ signaling

2. Type 1 ryanodine receptor in cardiac mitochondria: Transducer of excitation–metabolism coupling

3. Structural evidence for direct interaction between the molecular components of the transverse tubule/sarcoplasmic reticulum junction in skeletal muscle.

4. Multiple Diffusion Mechanisms Due to Nanostructuring in Crowded Environments

5. Active accumulation of Sr2+ by rat-liver mitochondria II. Competition between Ca2+ and Sr2+

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