Author:
Sanguinetti MC,Keating MT
Abstract
Cardiac action potentials are long because the magnitude of some potassium currents is reduced at depolarized potentials. The slow onset of repolarization is initiated by the opening of delayed rectifier potassium channels. A decrease in function of these channels resulting from gene mutations or pharmacological block increases risk of life-threatening ventricular arrhythmias.
Publisher
American Physiological Society
Cited by
3 articles.
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