Enhancement of L-type Ca2+ current from neonatal mouse ventricular myocytes by constitutively active PKC-βII
Author:
Affiliation:
1. Department of Physiology and Biophysics and
2. Section of Cardiology, Department of Medicine, College of Medicine, University of Illinois at Chicago, Chicago, Illinois 60607
Abstract
Publisher
American Physiological Society
Subject
Cell Biology,Physiology
Link
https://www.physiology.org/doi/pdf/10.1152/ajpcell.00494.2001
Reference24 articles.
1. Angiotensin II stimulates cardiac L-type Ca2+current by a Ca2+- and protein kinase C-dependent mechanism
2. PKC-independent inhibition of cardiac L-type Ca2+ channel current by phorbol esters
3. Tyrosine kinase and protein kinase C regulate L-type Ca2+ current cooperatively in human atrial myocytes
4. Protein kinase C regulation of cardiac calcium channels expressed in Xenopus oocytes
5. Expression of protein kinase C beta in the heart causes hypertrophy in adult mice and sudden death in neonates.
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