Influence of intracellular Ca2+ and alternative splicing on the pharmacological profile of ANO1 channels
Author:
Affiliation:
1. Department of Physiology and Cell Biology, University of Nevada School of Medicine, Reno, Nevada; and
2. Department of Pharmacology, Center for Cardiovascular Research, University of Nevada School of Medicine, Reno, Nevada
Abstract
Funder
HHS | NIH | National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK)
Research Enhancement Grant from the University of Nevada
Publisher
American Physiological Society
Subject
Cell Biology,Physiology
Link
https://www.physiology.org/doi/pdf/10.1152/ajpcell.00070.2016
Reference52 articles.
1. Mechanism of the Inhibition of Ca2+-Activated Cl− Currents by Phosphorylation in Pulmonary Arterial Smooth Muscle Cells
2. Activation of calcium-dependent chloride channels in rat parotid acinar cells.
3. Variomics Screen Identifies the Re-entrant Loop of the Calcium-activated Chloride Channel ANO1 That Facilitates Channel Activation
4. Pharmacological characterization of TMEM16A currents
5. X-ray structure of a calcium-activated TMEM16 lipid scramblase
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