Mitochondrial uncoupling in skeletal muscle by UCP1 augments energy expenditure and glutathione content while mitigating ROS production
Author:
Affiliation:
1. Department of Biochemistry, Microbiology and Immunology, Faculty of Medicine, University of Ottawa, Ottawa, Ontario, Canada; and
2. University of Ottawa Heart Institute, Ottawa, Ontario Canada
Abstract
Publisher
American Physiological Society
Subject
Physiology (medical),Physiology,Endocrinology, Diabetes and Metabolism
Link
https://www.physiology.org/doi/pdf/10.1152/ajpendo.00057.2013
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