CXCR3-independent role of CXCL10 in alveolar epithelial repair-Reference-Cited by-同舟云学术

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CXCR3-independent role of CXCL10 in alveolar epithelial repair

Published:2024-08-01 Issue:2 Volume:327 Page:L160-L172
ISSN:1040-0605
Container-title:American Journal of Physiology-Lung Cellular and Molecular Physiology
language:en
Short-container-title:American Journal of Physiology-Lung Cellular and Molecular Physiology

Author:

Zhang Yanli¹²^ORCID,Liang Jiurong²,Ye Jun¹^ORCID,Liu Ningshan²,Noble Paul W.²,Jiang Dianhua²

Affiliation:

1. State Key Laboratory of Common Mechanism Research for Major Diseases, Institute of Basic Medical Sciences, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, People’s Republic of China

2. Department of Medicine, Cedars-Sinai Medical Center, Los Angeles, California, United States

Abstract

CXCL10 may aid in lung injury recovery by promoting the proliferation of alveolar stem cells and using a distinct regulatory pathway from the classical one.

Funder

HHS | National Institutes of Health

MOST | National Natural Science Foundation of China

Publisher

American Physiological Society

Link

https://journals.physiology.org/doi/pdf/10.1152/ajplung.00301.2023

Reference49 articles.

1. Chemokines — Chemotactic Cytokines That Mediate Inflammation

2. Chemokine receptor specific for IP10 and mig: structure, function, and expression in activated T-lymphocytes.

3. IFN-γ-Inducible Protein 10 (IP-10; CXCL10)-Deficient Mice Reveal a Role for IP-10 in Effector T Cell Generation and Trafficking

4. CXCL10 Can Inhibit Endothelial Cell Proliferation Independently of CXCR3

5. Inhibition of pulmonary fibrosis in mice by CXCL10 requires glycosaminoglycan binding and syndecan-4

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