Disruption of the SP-A/SP-R210<sub>L</sub> (MYO18Aα) pathway prolongs gestation and reduces fetal survival during lipopolysaccharide-induced parturition in late gestation-Reference-Cited by-同舟云学术

Disruption of the SP-A/SP-R210_L (MYO18Aα) pathway prolongs gestation and reduces fetal survival during lipopolysaccharide-induced parturition in late gestation

Published:2024-04-01 Issue:4 Volume:326 Page:L508-L513
ISSN:1040-0605
Container-title:American Journal of Physiology-Lung Cellular and Molecular Physiology
language:en
Short-container-title:American Journal of Physiology-Lung Cellular and Molecular Physiology

Author:

Guan Zhiwei¹,Worth Brandon¹,Umstead Todd M.¹,Amatya Shaili¹,Booth Jennifer²,Chroneos Zissis C.¹³^ORCID

Affiliation:

1. Division of Neonatal-Perinatal Medicine, Pulmonary Immunology and Physiology Laboratory, Department of Pediatrics, Pennsylvania State University College of Medicine, Hershey, Pennsylvania, United States

2. Department of Comparative Medicine, Pennsylvania State University College of Medicine, Hershey, Pennsylvania, United States

3. Department of Microbiology and Immunology, Pennsylvania State University College of Medicine, Hershey, Pennsylvania, United States

Abstract

To our knowledge, this study is the first to report that SP-A prevents delay of labor and inflammation-induced stillbirth through the receptor SP-R210L.

Funder

Velter Foundation

Children's Miracle Network Hospitals

HHS | NIH | NHLBI | Division of Intramural Research

PSU | Penn State College of Medicine

Publisher

American Physiological Society

Link

https://journals.physiology.org/doi/pdf/10.1152/ajplung.00383.2023

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5. Multifactorial Regulation of Myometrial Contractility During Pregnancy and Parturition

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