Dysregulated NF-κB activation in cystic fibrosis: evidence for a primary inflammatory disorder
Author:
Affiliation:
1. Center for Lung Research, Vanderbilt University School of Medicine, Nashville, Tennessee 37232-2650
Publisher
American Physiological Society
Subject
Cell Biology,Physiology (medical),Pulmonary and Respiratory Medicine,Physiology
Link
https://www.physiology.org/doi/pdf/10.1152/ajplung.2001.281.1.L69
Reference5 articles.
1. Activation of NF-kappaB by adherent Pseudomonas aeruginosa in normal and cystic fibrosis respiratory epithelial cells.
2. Role of unphosphorylated, newly synthesized I kappa B beta in persistent activation of NF-kappa B
3. High Susceptibility for Cystic Fibrosis Human Airway Gland Cells to Produce IL-8 Through the IκB Kinase α Pathway in Response to Extracellular NaCl Content
4. Exaggerated Activation of Nuclear Factor- κ B and Altered I κ B- β Processing in Cystic Fibrosis Bronchial Epithelial Cells
5. Activation of NF-κB in airway epithelial cells is dependent on CFTR trafficking and Cl− channel function
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2. Myeloid arginase-1 controls excessive inflammation and modulates T cell responses in Pseudomonas aeruginosa pneumonia;Immunobiology;2021-01
3. Toll like Receptor signalling by Prevotella histicola activates alternative NF-κB signalling in Cystic Fibrosis bronchial epithelial cells compared to P. aeruginosa;PLOS ONE;2020-10-08
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5. Pulmonary Disease in Cystic Fibrosis;Kendig's Disorders of the Respiratory Tract in Children;2019
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