Preconditioning protects by inhibiting the mitochondrial permeability transition-Reference-Cited by-同舟云学术

Preconditioning protects by inhibiting the mitochondrial permeability transition

Published:2004-08 Issue:2 Volume:287 Page:H841-H849
ISSN:0363-6135
Container-title:American Journal of Physiology-Heart and Circulatory Physiology
language:en
Short-container-title:American Journal of Physiology-Heart and Circulatory Physiology

Author:

Hausenloy Derek J.,Yellon Derek M.,Mani-Babu Siva,Duchen Michael R.

Publisher

American Physiological Society

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology

Link

http://www.physiology.org/doi/pdf/10.1152/ajpheart.00678.2003

Reference48 articles.

1. Mitochondrial ATP-Sensitive Potassium Channels Inhibit Apoptosis Induced by Oxidative Stress in Cardiac Cells

2. Fluorescent cationic probes of mitochondria. Metrics and mechanism of interaction

3. Mitochondrial KATP channel opening protects a human atrial-derived cell line by a mechanism involving free radical generation

4. Kinetic evidence for a heart mitochondrial pore activated by Ca2+, inorganic phosphate and oxidative stress. A potential mechanism for mitochondrial dysfunction during cellular Ca2+ overload

5. Crompton M, Ellinger H, and Costi A.Inhibition by cyclosporin A of a Ca2+-dependent pore in heart mitochondria activated by inorganic phosphate and oxidative stress.Biochem J255: 357–360, 1988.

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