Sexual dimorphism in doxorubicin cardiotoxicity: two sides of a complex coin
Author:
Affiliation:
1. IRCCS Ospedale Policlinico San Martino, Genova, Italy
2. Department of Internal Medicine, University of Genova, Genova, Italy
Publisher
American Physiological Society
Subject
Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology
Link
https://journals.physiology.org/doi/pdf/10.1152/ajpheart.00566.2023
Reference9 articles.
1. Cancer Therapy–Induced Cardiotoxicity: Basic Mechanisms and Potential Cardioprotective Therapies
2. p38 MAPK Pathway in the Heart: New Insights in Health and Disease
3. Anthracycline cardiotoxicity is exacerbated by global p38β genetic ablation in a sexually dimorphic manner but unaltered by cardiomyocyte-specific p38α loss
4. Improving translational research in sex-specific effects of comorbidities and risk factors in ischaemic heart disease and cardioprotection: position paper and recommendations of the ESC Working Group on Cellular Biology of the Heart
5. Testosterone Antagonizes Doxorubicin‐Induced Senescence of Cardiomyocytes
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