Contribution of TRPV1 Receptor^|^ndash;Expressing Fibers to Spinal Ventral Root After-Discharges and Mechanical Hyperalgesia in a Spared Nerve Injury (SNI) Rat Model-Reference-Cited by-同舟云学术

Contribution of TRPV1 Receptor^|^ndash;Expressing Fibers to Spinal Ventral Root After-Discharges and Mechanical Hyperalgesia in a Spared Nerve Injury (SNI) Rat Model

Published:2013 Issue:1 Volume:121 Page:9-16
ISSN:1347-8613
Container-title:Journal of Pharmacological Sciences
language:en
Short-container-title:J Pharmacol Sci

Author:

Yamamoto Shohei,Ohsawa Masahiro,Ono Hideki

Publisher

Japanese Pharmacological Society

Subject

Pharmacology,Molecular Medicine

Reference27 articles.

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2. A novel behavioral model of neuropathic pain disorders produced in rats by partial sciatic nerve injury

3. An experimental model for peripheral neuropathy produced by segmental spinal nerve ligation in the rat

4. Spared nerve injury: an animal model of persistent peripheral neuropathic pain

5. 6 Yamamoto S, Honda M, Tanabe M, Ono H. Spinal ventral root after-discharges as a pain index: involvement of NK-1 and NMDA receptors. Brain Res. 2006;1082:115–123.

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