Effects of Lactate on Intracellular pH and Hypercontracture During Simulated Ischemia and Reperfusion in Cardiac Ventricular Myocytes of the Guinea Pig
Author:
Publisher
Elsevier BV
Subject
Pharmacology
Link
https://www.jstage.jst.go.jp/article/jjp/80/4/80_4_343/_pdf
Reference31 articles.
1. Protons in ischemia: where do they come from; where do they go?;Dennis;J Mol Cell Cardiol,1991
2. Is lactate-induced myocardial ischaemic injury mediated by decreased pH or increased intracellular lactate?;Cross;J Mol Cell Cardiol,1995
3. Lactate does not enhance anoxia/reoxygenation damages in adult rat cardiac myocytes;Geisbuhler;J Mol Cell Cardiol,1990
4. Reducing lactate accumulation does not attenuate lethal ischemic injury in isolated perfused rat hearts;Vander Heide;Am J Physiol,1996
5. Effect of stepwise normalization of perfusate pH on postischemic functional recovery and Ca overload in isolated rat hearts;Ebihara;Jpn Circ J,1996
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1. Absence of glucose transporter 4 diminishes electrical activity of mouse hearts during hypoxia;Experimental Physiology;2013-01-11
2. Mechanism of glutathione depletion during simulated ischemia-reperfusion of H9c2 cardiac myocytes;Free Radical Research;2011-07-14
3. Regional Electroporation of Single Cardiac Myocytes in a Focused Electric Field;Analytical Chemistry;2009-12-18
4. Protective Effect of Benidipine Against the Abnormal Electrical Activity in Single Ventricular Myocytes of the Guinea Pig Under Simulated Ischemic Conditions and Reperfusion;Japanese Journal of Pharmacology;2000
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