Amyloid β Peptide Specifically Promotes Phosphorylation and Nuclear Translocation of the Extracellular Signal-Regulated Kinase in Cultured Rat Cortical Astrocytes
Author:
Publisher
Japanese Pharmacological Society
Subject
Pharmacology,Molecular Medicine
Reference24 articles.
1. Neurotrophic and Neurotoxic Effects of Amyloid β Protein: Reversal by Tachykinin Neuropeptides
2. In vitro aging of ß-amyloid protein causes peptide aggregation and neurotoxicity
3. Calcium-destabilizing and neurodegenerative effects of aggregated β-amyloid peptide are attenuated by basic FGF
4. Hydrogen peroxide mediates amyloid β protein toxicity
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1. Natural Intracellular Consequences of Amyloid;Intracellular Consequences of Amyloid in Alzheimer's Disease;2016
2. Amyloid-β as a Modulator of Synaptic Plasticity;Journal of Alzheimer's Disease;2010-11-25
3. Oral supplementation with Leu-Ile, a hydrophobic dipeptide, prevents the impairment of memory induced by amyloid beta in mice via restraining the hyperphosphorylation of extracellular signal-regulated kinase;Behavioural Brain Research;2010-07
4. Early diagnostic accuracy and pathophysiologic relevance of an autopsy-confirmed Alzheimer's disease peripheral biomarker;Neurobiology of Aging;2010-06
5. Astaxanthine Secured Apoptotic Death of PC12 Cells Induced by β-Amyloid Peptide 25–35: Its Molecular Action Targets;Journal of Medicinal Food;2010-06
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