Inhibition of Agonist-Induced Positive Inotropy by a Selective Rho-Associated Kinase Inhibitor, Y-27632
Author:
Publisher
Japanese Pharmacological Society
Subject
Pharmacology,Molecular Medicine
Reference15 articles.
1. The Role of Phosphoinositide Hydrolysis in the Regulation of Cardiac Function via α-Adrenergic, Endothelin, and Angiotensin Receptors
2. Endothelin activates voltage-dependent Ca2+ current by a G protein-dependent mechanism in rabbit cardiac myocytes.
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2. Endothelin-1 and angiotensin II modulate rate and contraction amplitude in a subpopulation of mouse embryonic stem cell-derived cardiomyocyte-containing bodies;Stem Cell Research;2011-01
3. Activation of muscarinic receptors elicits inotropic responses in ventricular muscle from rats with heart failure through myosin light chain phosphorylation;British Journal of Pharmacology;2009-02
4. Targeting cerebrovascular Rho-kinase in stroke;Expert Opinion on Therapeutic Targets;2008-11-13
5. Reduced contractile response to α1-adrenergic stimulation in atria from mice with chronic cardiac calmodulin kinase II inhibition;Journal of Molecular and Cellular Cardiology;2007-03
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