Ranolazine Attenuates the Enhanced Reverse Na+-Ca2+ Exchange Current via Inhibiting Hypoxia-Increased Late Sodium Current in Ventricular Myocytes
Author:
Publisher
Japanese Pharmacological Society
Subject
Pharmacology,Molecular Medicine
Link
https://www.jstage.jst.go.jp/article/jphs/124/3/124_13202FP/_pdf
Reference49 articles.
1. 1 Kiyosue T, Arita M. Late sodium current and its contribution to action potential configuration in guinea pig ventricular myocytes. Circ Res. 1989;64:389–397.
2. 2 Qian C, Ma J, Zhang P, Luo A, Wang C, Ren Z, et al. Resveratrol attenuates the Na(+)-dependent intracellular Ca(2+) overload by inhibiting H(2)O(2)-induced increase in late sodium current in ventricular myocytes. PloS One. 2012;7:e51358.
3. 3 Zheng J, Ma J, Zhang P, Hu L, Fan X, Tang Q. Milrinone inhibits hypoxia or hydrogen dioxide-induced persistent sodium current in ventricular myocytes. Eur J Pharmacol. 2009;616:206–212.
4. 4 Zhang S, Ma JH, Zhang PH, Luo AT, Ren ZQ, Kong LH. Sophocarpine attenuates the Na(+)-dependent Ca(2+) overload induced by Anemonia sulcata toxin-increased late sodium current in rabbit ventricular myocytes. J Cardiovasc Pharmacol. 2012;60:357–366.
5. 5 Antzelevitch C. Electrical heterogeneity, cardiac arrhythmias, and the sodium channel. Circ Res. 2000;87:964–965.
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