Moderate cold exposure contributes to the extension of lifespan by alleviating the aggregation of disease-related proteins
Author:
Affiliation:
1. Department of Medicine (Section of Cardiovascular Research) , Baylor College of Medicine , Houston , USA
Publisher
Walter de Gruyter GmbH
Link
https://www.sciendo.com/pdf/10.2478/fzm-2023-0017
Reference9 articles.
1. Lee H J, Alirzayeva H, Koyuncu S, et al. Cold temperature extends longevity and prevents disease-related protein aggregation through PA28γ-induced proteasomes. Nat Aging, 2023; 3(5): 546–566.
2. Cold exposure and winter mortality from ischaemic heart disease, cerebrovascular disease, respiratory disease, and all causes in warm and cold regions of Europe. The Eurowinter Group. Lancet, 1997; 349(9062): 1341–1346.
3. Al-Kindi S, Motairek I, Khraishah H, et al. Cardiovascular disease burden attributable to non-optimal temperature: analysis of the 1990–2019 global burden of disease. Eur J Prev Cardiol, 2023: zwad130.
4. Sugimoto S, Mena H A, Sansbury B E, et al. Brown adipose tissue-derived MaR2 contributes to cold-induced resolution of inflammation. Nat Metab, 2022; 4(6): 775–790.
5. Li C, Sun X N, Zhao S, et al. Crosstalk between adipose tissue and the heart: an update. J Transl Int Med, 2022; 10(3): 219–226.
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