Synovial fibroblasts integrate inflammatory and neuroendocrine stimuli to drive rheumatoid arthritis
Author:
Publisher
Informa UK Limited
Subject
Immunology,Immunology and Allergy
Link
http://www.tandfonline.com/doi/pdf/10.1586/1744666X.2015.1066674
Reference22 articles.
1. Innate immunity triggers IL-32 expression by fibroblast-like synoviocytes in rheumatoid arthritis
2. TNF-α confers resistance to Fas-mediated apoptosis in rheumatoid arthritis through the induction of soluble Fas
3. Anchorage on fibronectin via VLA-5 ( 5 1 integrin) protects rheumatoid synovial cells from Fas-induced apoptosis
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