Animal models for autosomal dominant frontal lobe epilepsy: on the origin of seizures
Author:
Publisher
Informa UK Limited
Subject
Pharmacology (medical),Neurology (clinical),General Neuroscience
Link
http://www.tandfonline.com/doi/pdf/10.1586/ern.10.130
Reference54 articles.
1. A missense mutation in the neuronal nicotinic acetylcholine receptor α4 subunit is associated with autosomal dominant nocturnal frontal lobe epilepsy
2. A novel mutation of CHRNA4 responsible for autosomal dominant nocturnal frontal lobe epilepsy
3. An Insertion Mutation of the CHRNA4 Gene in a Family With Autosomal Dominant Nocturnal Frontal Lobe Epilepsy
4. A New Chrna4 Mutation with Low Penetrance in Nocturnal Frontal Lobe Epilepsy
5. A novel mutation of the nicotinic acetylcholine receptor gene CHRNA4 in sporadic nocturnal frontal lobe epilepsy
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3. Sleep-related hypermotor epilepsy associated mutations uncover important kinetic roles of α4β2- nicotinic acetylcholine receptor intracellular structures;PLOS ONE;2021-03-03
4. Upregulated and Hyperactivated Thalamic Connexin 43 Plays Important Roles in Pathomechanisms of Cognitive Impairment and Seizure of Autosomal Dominant Sleep-Related Hypermotor Epilepsy with S284L-Mutant α4 Subunit of Nicotinic ACh Receptor;Pharmaceuticals;2020-05-18
5. Upregulated Connexin 43 Induced by Loss-of-Functional S284L-Mutant α4 Subunit of Nicotinic ACh Receptor Contributes to Pathomechanisms of Autosomal Dominant Sleep-Related Hypermotor Epilepsy;Pharmaceuticals;2020-03-29
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