The Role of Sestrins in the Regulation of the Cellular Response to Stress
-
Published:2022-07-20
Issue:4
Volume:12
Page:347-364
-
ISSN:2079-0864
-
Container-title:Biology Bulletin Reviews
-
language:en
-
Short-container-title:Biol Bull Rev
Publisher
Pleiades Publishing Ltd
Subject
Automotive Engineering
Reference127 articles.
1. Abe, T., Makino, N., Furukawa, T., et al., Identification of three commonly deleted regions on chromosome arm 6q in human pancreatic cancer, Genes, Chromosomes Cancer, 1999, vol. 25, no. 1, pp. 60–64. 2. Andrysik, Z., Galbraith, M.D., Guarnieri, A.L., et al., Identification of a core TP53 transcriptional program with highly distributed tumor suppressive activity, Genome Res., 2017, vol. 27, no. 10, pp. 1645–1657. 3. Bae, S.H., Sung, S.H., Oh, S.Y., et al., Sestrins activate Nrf2 by promoting p62-dependent autophagic degradation of Keap1 and prevent oxidative liver damage, Cell Metab., 2013, vol. 17, no. 1, pp. 73–84. 4. Bar-Peled, L., Chantranupong, L., Cherniack, A.D., et al., A tumor suppressor complex with GAP activity for the Rag GTPases that signal amino acid sufficiency to mTORC1, Science, 2013, vol. 340, no. 6136, pp. 1100–1106. 5. Ben-Sahra, I., Dirat, B., Laurent, K., et al., Sestrin2 integrates Akt and mTOR signaling to protect cells against energetic stress-induced death, Cell Death Differ., 2013, vol. 20, no. 4, pp. 611–619.
|
|