Author:
Igudin E. L.,Spirin P. V.,Prasolov V. S.,Zubkova N. A.,Petryaikina E. E.,Tyul’pakov A. N.,Rubtsov P. M.
Subject
Structural Biology,Biophysics
Reference20 articles.
1. Slingerland A.S. 2006. Monogenic diabetes in children and young adults: Challenges for researcher, clinician and patient. Rev. Endocr. Metab. Disord. 7, 171–185.
2. Vaxillaire M., Froguel P. 2008. Monogenic diabetes in the young, pharmacogenetics and relevance to multi-factorial forms of type 2 diabetes. Endocr. Rev. 29, 254–264.
3. Ellard S., Beards F., Allen L.I., Shepherd M., Ballantyne E., Harvey R., Hattersley A.T. 2000. A high prevalence of glucokinase mutations in gestational diabetic subjects selected by clinical criteria. Diabetologia. 43, 250–253.
4. Massa O., Meschi F., Cuesta-Munoz A., et al. 2001. High prevalence of glucokinase mutations in Italian children with MODY: Influence on glucose tolerance, first-phase insulin response, insulin sensitivity and BMI. Diabetes Study Group of the Italian Society of Paediatric Endocrinology and Diabetes (SIEDP). Diabetologia. 44, 898–905.
5. Estalella I., Rica I., Perez de Nanclares G., et al. 2007. Mutations in GCK and HNF-1alpha explain the majority of cases with clinical diagnosis of MODY in Spain. Clin. Endocrinol. (Oxford). 67, 538–546.
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