Genetically modified animals as models of the pathological processes in psoriasis
Author:
Publisher
Pleiades Publishing Ltd
Subject
Structural Biology,Biophysics
Link
http://link.springer.com/content/pdf/10.1134/S0026893314040153.pdf
Reference96 articles.
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4. Gallardo Torres H.I., Gijbels M.J., HegnEsch H., Kraal G. 1995. Chronic proliferative dermatitis in mice: neutrophil-endothelium interactions and the role of adhesion molecules. Pathobiology. 63(6), 341–347.
5. Ikeda F., Deribe Y.L., Skånland S.S., Stieglitz B., Grabbe C., Franz-Wachtel M., van Wijk S.J., Goswami P., Nagy V., Terzic J., Tokunaga F., Androulidaki A., Nakagawa T., Pasparakis M., Iwai K., Sundberg J.P., Schaefer L., Rittinger K., Macek B., Dikic I. 2011. SHARPIN forms a linear ubiquitin ligase complex regulating NF-kappaB activity and apoptosis. Nature. 471(7340), 637–641.
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1. Past, present and future of in vitro 3D reconstructed inflammatory skin models to study psoriasis;Experimental Dermatology;2018-04-06
2. Acitretin and aloe-emodin loaded chitin nanogel for the treatment of psoriasis;European Journal of Pharmaceutics and Biopharmaceutics;2016-10
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