Fifity years after the identification of the cause of SMON-Reference-Cited by-同舟云学术

Fifity years after the identification of the cause of SMON

Published:2021 Issue:2 Volume:61 Page:109-114
ISSN:0009-918X
Container-title:Rinsho Shinkeigaku
language:en
Short-container-title:Rinsho Shinkeigaku

Author:

Kuru Satoshi¹

Affiliation:

1. Department of Neurology, National Hospital Organization Suzuka Hospital

Publisher

Societas Neurologica Japonica

Subject

Neurology (clinical)

Link

https://www.jstage.jst.go.jp/article/clinicalneurol/61/2/61_cn-001500/_pdf

Reference14 articles.

1. 1) Sobue I. Clinical aspects of subacute myelo-optico-neuropathy (SMON). In: Vinken PJ, Bruyn GW, Cohen MM, et al., eds. Intoxications of the nervous system Part 2. Handbook of clinical neurology, vol 37. Amsterdam, North-Holland, 1979, p.113-115.

2. 2) Konagaya M, Matsumoto A, Takase S, et al. Clinical analysis of longstanding subacute myelo-optico-neuropathy: sequelae of clioquinol at 32 years after its ban. J Neurol Sci 2004;218:85-90.

3. 15) Tateishi J, Kuroda S, Saito A, et al. Myelo-optic neuropathy induced by clioquinol in animals. Lancet 1971;2:1263-1264.

4. 24) Andersson DA, Gentry C, Moss S, et al. Clioquinol and pyrithione activate TRPA1 by increasing intracellular Zn²⁺. Proc Natl Acad Sci U S A 2009;106:8374-8379.

5. 25) Katsuyama M, Ibi M, Matsumoto M, et al. Clioquinol increases the expression of VGF, a neuropeptide precursor, through induction of c-Fos expression. J Pharmacol Sci 2014;124:427-432.

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