Thalassemia Incidentally Found by Marked Erythrocytosis Due to an Ochre Mutation at Codon 35 in a Japanese Man.-Reference-Cited by-同舟云学术

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Thalassemia Incidentally Found by Marked Erythrocytosis Due to an Ochre Mutation at Codon 35 in a Japanese Man.

Published:1995 Issue:12 Volume:34 Page:1198-1200
ISSN:0918-2918
Container-title:Internal Medicine
language:en
Short-container-title:Intern. Med.

Author:

YASUNAGA Mutsumi¹,FUJIYAMA Yoshihide¹,MIYAGAWA Akiko¹,INOUE Tetsuya¹,ANDOH Akira¹,HODOHARA Keiko¹,BAMBA Tadao¹,INOUE Takafumi²,YAWATA Yoshihito²,HARANO Keiko³,HARANO Teruo³

Affiliation:

1. the Second Department of Internal Medicine, Shiga University of Medical Science

2. the Departments of Hematology, Kawasaki Medical School

3. the Departments of Biochemistry, Kawasaki Medical School

Publisher

Japanese Society of Internal Medicine

Subject

General Medicine,Internal Medicine

Link

http://www.jstage.jst.go.jp/article/internalmedicine1992/34/12/34_12_1198/_pdf

Cited by 3 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Polycythemia Vera and the Jak2(V617F) Mutation in a Case of Hereditary Spherocytosis;The American Journal of the Medical Sciences;2013-10

2. LETTER TO THE EDITOR: A novel homozygous point mutation at codon 82 (HBB:c.247A > T) in the beta-globin gene leads to thalassemia major;International Journal of Laboratory Hematology;2010-08-23

3. A novel beta-globin mutation (HBB:c.107A>G; or codon 35 β (A→G)) at alpha–beta chain interfaces;Annals of Hematology;2009-06-02

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