Treatment of Mitochondrial Diabetes with a Peroxisome Proliferator-activated Receptor (PPAR)-gamma Agonist
Author:
Affiliation:
1. Department of Metabolic Medicine, Graduate School of Medicine, Osaka University, Japan
2. Department of Metabolism and Atherosclerosis, Graduate School of Medicine, Osaka University, Japan
Publisher
Japanese Society of Internal Medicine
Subject
General Medicine,Internal Medicine
Link
https://www.jstage.jst.go.jp/article/internalmedicine/55/9/55_55.4418/_pdf
Reference37 articles.
1. 1. Odawara M, Sasaki K, Yamashita K. Prevalence and clinical characterization of Japanese diabetes mellitus with an A-to-G mutation at nucleotide 3243 of the mitochondrial tRNALeu(UUR) gene. J Clin Endocrinol Metab 80: 1290-1294, 1995.
2. 2. Kadowaki T, Kadowaki H, Mori Y, et al. A subtype of diabetes mellitus associated with a mutation of mitochondrial DNA. N Engl J Med 330: 962-968, 1994.
3. 3. Oka Y, Katagiri H, Ishihara H, Asano T, Kobayashi T, Kikuchi M. Beta-cell loss and glucose induced signalling defects in diabetes mellitus caused by mitochondrial tRNALeu(UUR) gene mutation. Diabet Med 13 (9 Suppl 6): S98-S102, 1996.
4. 4. Murphy R, Turnbull DM, Walker M, Hattersley AT. Clinical features, diagnosis and management of maternally inherited diabetes and deafness (MIDD) associated with the 3243A>G mitochondrial point mutation. Diabet Med 25: 383-399, 2008.
5. 5. Lynn S, Borthwick GM, Charnley RM, Walker M, Turnbull DM. Heteroplasmic ratio of the A3243G mitochondrial DNA mutation in single pancreatic beta cells. Diabetologia 46: 296-299, 2003.
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