Glucocorticoid-sensitive Paroxysmal Atrial Fibrillation, Sick Sinus Syndrome, and Mitral Regurgitation in a Patient with Malignant Rheumatoid Vasculitis
Author:
Affiliation:
1. Division of Cardiovascular Medicine, Department of Internal Medicine, Kobe University Graduate School of Medicine, Japan
2. Department of Rheumatology and Clinical Immunology, Kobe University Graduate School of Medicine, Japan
Publisher
Japanese Society of Internal Medicine
Subject
General Medicine,Internal Medicine
Link
https://www.jstage.jst.go.jp/article/internalmedicine/58/21/58_3090-19/_pdf
Reference6 articles.
1. 1. Lazzerini PE, Capecchi PL, Laghi-Pasini F. Systemic inflammation and arrhythmic risk: lessons from rheumatoid arthritis. Eur Heart J 38: 1717-1727, 2017.
2. 2. Guo Y, Lip GY, Apostolakis S. Inflammation in atrial fibrillation. J Am Coll Cardiol 60: 2263-2270, 2012.
3. 3. Japan Intractable Disease Information Center. [Malignant Rheumatoid Vasculitis (designated intractable disease 46)] [Internet]. [cited 2018 May 15]. Available from: http://www.nanbyou.or.jp/entry/205 (in Japanese)
4. 4. Liu L, Lee J, Fu G, et al. Activation of peripheral blood CD 3 (+) T-lymphocytes in patients with atrial fibrillation. Int Heart J 53: 221-224, 2012.
5. 5. Sugiura A, Funabashi N, Ozawa K, Kobayashi Y. Immunological and inflammatory processes in systemic autoimmune disease may not only cause pericardium inflammation, but may also cause mitral valve deterioration and left ventricular wall thickening. Int J Cardiol 215: 466-471, 2016.
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