Leucine-rich Glioma-inactivated 1 Encephalitis Followed by Isaacs Syndrome: Alternating Presence of Pathogenic Autoantibodies to Leucine-rich Glioma-inactivated 1 and Contactin-associated Protein-like 2
Author:
Affiliation:
1. Department of Neurology, Tokyo Metropolitan Neurological Hospital, Japan
2. Division of Neurology, Department of Medicine, Nihon University School of Medicine, Japan
Publisher
Japanese Society of Internal Medicine
Subject
General Medicine,Internal Medicine
Link
https://www.jstage.jst.go.jp/article/internalmedicine/advpub/0/advpub_9670-22/_pdf
Reference12 articles.
1. 1. Irani SR, Alexander S, Waters P, et al. Antibodies to Kv1 potassium channel-complex proteins leucine-rich, glioma inactivated 1 protein and contactin-associated protein-2 in limbic encephalitis, Morvan's syndrome and acquired neuromyotonia. Brain 133: 2734-2748, 2010.
2. 2. Binks SNM, Klein CJ, Waters P, Pittock SJ, Irani SR. LGI1, CASPR2 and related antibodies: a molecular evolution of the phenotypes. J Neurol Neurosurg Psychiatry 89: 526-534, 2018.
3. 3. van Sonderen A, Ariño H, Petit-Pedrol M, et al. The clinical spectrum of Caspr2 antibody-associated disease. Neurology 87: 521-528, 2016.
4. 4. Graus F, Titulaer MJ, Balu R, et al. A clinical approach to diagnosis of autoimmune encephalitis. Lancet Neurol 15: 391-404, 2016.
5. 5. van Sonderen A, Mar Petit-Pedrol, Josep Dalmau, Maarten JTitulaer. The value of LGI1, Caspr2 and voltage-gated potassium channelantibodies in encephalitis. Nature Reviews Neurology 13: 290-301, 2017.
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