Tumor-induced Osteomalacia Caused by a Parotid Tumor
Author:
Affiliation:
1. Division of Nephrology and Endocrinology, The University of Tokyo Hospital, Japan
2. Department of Otolaryngology, The University of Tokyo Hospital, Japan
3. Department of Pathology, The University of Tokyo Hospital, Japan
Publisher
Japanese Society of Internal Medicine
Subject
General Medicine,Internal Medicine
Link
https://www.jstage.jst.go.jp/article/internalmedicine/56/5/56_56.7565/_pdf
Reference15 articles.
1. 1. Shimada T, Mizutani S, Muto T, et al. Cloning and characterization of FGF23 as a causative factor of tumor-induced osteomalacia. Proc Natl Acad Sci U S A 98: 6500-6505, 2001.
2. 2. Ito N, Findlay DM, Atkins GJ. Osteocyte communication with the kidney via the production of FGF23: remote control of phosphate homeostasis. Clin Rev Bone Miner Metab 12: 44-58, 2014.
3. 3. Yamazaki Y, Okazaki R, Shibata M, et al. Increased circulatory level of biologically active full-length FGF-23 in patients with hypophosphatemic rickets/osteomalacia. J Clin Endocrinol Metab 87: 4957-4960, 2002.
4. 4. Weidner N, Santa Cruz D. Phosphaturic mesenchymal tumors. A polymorphous group causing osteomalacia or rickets. Cancer 59: 1442-1454, 1987.
5. 5. Uno T, Kawai K, Kunii N, et al. Osteomalacia caused by skull base tumors: report of 2 cases. Neurosurgery 69: E239-E244; discussion E44, 2011.
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