Before SARS-CoV-2-related Encephalitis Can Be Attributed to Anti-GFAP Antibodies, Alternative Etiologies Must Be Ruled out
Author:
Affiliation:
1. Neurology & Neurophysiology Center, Austria
Publisher
Japanese Society of Internal Medicine
Link
https://www.jstage.jst.go.jp/article/internalmedicine/63/7/63_3237-23/_pdf
Reference4 articles.
1. 1. Tajiri M, Takasone K, Kodaira M, Kimura A, Shimohata T, Sekijima Y. Autoimmune glial fibrillary acidic protein astrocytopathy following SARS-CoV-2 infection. Intern Med 63: 337-339, 2024.
2. 2. Leineweber TD, Ghathian K, Lisby JG, et al. Evaluation of four laboratory-based high-throughput SARS-CoV-2 automated antigen tests compared to RT-PCR on nasal and oropharyngeal samples. J Clin Virol 164: 105472, 2023.
3. 3. Pilotto A, Masciocchi S, Volonghi I, et al. Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) encephalitis is a cytokine release syndrome: evidences from cerebrospinal fluid analyses. Clin Infect Dis 73: e3019-e3026, 2021.
4. 4. Tsagkaris C, Bilal M, Aktar I, et al. Cytokine storm and neuropathological alterations in patients with neurological manifestations of COVID-19. Curr Alzheimer Res 19: 641-657, 2022.
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