Coping Mechanisms against Oxidative Stress in Kidney Disease
Author:
Affiliation:
1. Division of Nephrology and Hypertension, Department of Internal Medicine, Kawasaki Medical School
Publisher
Japanese Society of Internal Medicine
Subject
General Medicine
Link
https://www.jstage.jst.go.jp/article/naika/106/7/106_1473/_pdf
Reference10 articles.
1. 1) Matsuguma K, et al: Molecular mechanism for elevation of asymmetric dimethylarginine and its role for hypertension in chronic kidney disease. J Am Soc Nephrol 17: 2176-2183, 2006.
2. 2) Deen WM: What determines glomerular capillary permeability? J Clin Invest 114: 1412-1414, 2004.
3. 3) Kuwabara A, et al: Deterioration of glomerular endothelial surface layer induced by oxidative stress is implicated in altered permeability of macromolecules in Zucker fatty rats. Diabetologia 53: 2056-2065, 2010.
4. 4) Li JM, Shah AM: ROS generation by nonphagocytic NADPH oxidase: potential relevance in diabetic nephropathy. J Am Soc Nephrol 14: S221-226, 2003.
5. 5) Fujimoto S, et al: Olmesartan ameliorates progressive glomerular injury in subtotal nephrectomized rats through suppression of superoxide production. Hypertens Res 31: 305-313, 2008.
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