Store operated calcium entry in NIH-3T3 cells
Author:
Affiliation:
1. Nihon University School of Dentistry at Matsudo
2. National Institute of Environmental Health Sciences-NIH
Publisher
University of Tokushima Faculty of Medicine
Subject
General Biochemistry, Genetics and Molecular Biology,General Medicine
Link
http://www.jstage.jst.go.jp/article/jmi/56/Supplement/56_Supplement_381/_pdf
Reference11 articles.
1. A model for receptor-regulated calcium entry
2. A point mutation at the ATP-binding site of the EGF-receptor abolishes signal transduction.
3. Evidence that type II insulin-like growth factor receptor is coupled to calcium gating system
4. Ionic events induced by epidermal growth factor. Evidence that hyperpolarization and stimulated cation influx play a role in the stimulation of cell growth.
5. P2Y purinoceptors in normal NIH 3T3 and in NIH 3T3 overexpressing c-ras
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