Expression of NADPH oxidase and production of reactive oxygen species contribute to ureteric bud branching and nephrogenesis
Author:
Affiliation:
1. Department of Pediatrics, Institute of Biomedical Sciences, Tokushima University Graduate School, Tokushima, Japan
Funder
Japan Society for the Promotion of Science
Publisher
University of Tokushima Faculty of Medicine
Subject
General Biochemistry, Genetics and Molecular Biology,General Medicine
Link
https://www.jstage.jst.go.jp/article/jmi/66/1.2/66_93/_pdf
Reference24 articles.
1. 1. Shah SV, Baliga R, Rajapurkar M, Fonseca VA : Oxidants inchronic kidney disease. J Am Soc Nephrol 18 : 16-28, 2007
2. 2. Lambeth JD : Nox enzymes, ROS, and chronic disease : anexample of antagonistic pleiotropy. Free Radic Biol Med 43 :332-347, 2007
3. 3. Chen S,Meng XF, Zhang C : Role of NADPH oxidase-mediatedreactive oxygen species in podocyte injury. Biomed Res Int2013 : 839761, 2013
4. 4. Barnes JL, Gorin Y : Myofibroblast differentiation duringfibrosis : role of NAD(P)H oxidases. Kidney Int 79 : 944-956,2011
5. 5. Schreck C, O’Connor PM : NAD(P)H oxidase and renal epithelialion transport. Am J Physiol Regul Integr Comp Physiol300 : R1023-1029, 2011
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