Inhibition of Growth and Induction of Differentiation of Glioma Cell Lines by Oncostatin M (OSM)
Author:
Publisher
Informa UK Limited
Subject
Cell Biology,Clinical Biochemistry,Endocrinology
Link
http://www.tandfonline.com/doi/pdf/10.3109/08977199809117189
Reference33 articles.
1. Oncostatin-M stimulates tyrosine protein phosphorylation in parallel with the activation of p42MAPK/ERK-2 in Kaposi's cells. Evidence that this pathway is important in Kaposi cell growth.
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4. Oncostatin M
5. Single-step method of RNA isolation by acid guanidinium thiocyanate-phenol-chloroform extraction
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1. The clinical relevance of OSM in inflammatory diseases: a comprehensive review;Frontiers in Immunology;2023-09-29
2. Association of oncostatin M receptor polymorphisms with clinical recurrence of ovarian cancer in the Chinese Han population;Biomarkers in Medicine;2022-04
3. Hyperbaric oxygen suppresses stemness-associated properties and Nanog and oncostatin M expression, but upregulates β-catenin in orthotopic glioma models;Journal of International Medical Research;2019-12-09
4. Oncostatin M as a new diagnostic, prognostic and therapeutic target in inflammatory bowel disease (IBD);Expert Opinion on Therapeutic Targets;2019-10-15
5. Oncostatin M receptor, positively regulated by SP1, promotes gastric cancer growth and metastasis upon treatment with Oncostatin M;Gastric Cancer;2019-02-18
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