Hydrolysis of Glycosyl-Phosphatidylinositol in Response to Insulin is Reduced in Cells Bearing Kinase-Deficient Insulin Receptors
Author:
Affiliation:
1. Department of Molecular Biology, Memorial Sloan Kettering Cancer Center, 1275 York Avenue, NewYork, New York, 10021
2. Departamento de Metabolismo, Nutricion y Hormonas, Fundacion Jimenez Diaz, Avenida Reyes Catolicos 2, 28040, Madrid, Spain
Publisher
Informa UK Limited
Subject
Cell Biology,Clinical Biochemistry,Endocrinology
Link
https://www.tandfonline.com/doi/pdf/10.3109/08977199009071496
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4. Phosphorus Assay in Column Chromatography
5. Human insulin receptors mutated at the ATP-binding site lack protein tyrosine kinase activity and fail to mediate postreceptor effects of insulin.
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